High-dose folic acid improves endothelial function by increasing tetrahydrobiopterin and decreasing homocysteine levels

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Folic acid improves endothelial function in coronary artery disease via mechanisms largely independent of homocysteine lowering.

BACKGROUND Homocysteine is a risk factor for coronary artery disease (CAD), although a causal relation remains to be proven. The importance of determining direct causality rests in the fact that plasma homocysteine can be safely and inexpensively reduced by 25% with folic acid. This reduction is maximally achieved by doses of 0.4 mg/d. High-dose folic acid (5 mg/d) improves endothelial function...

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Largely Independent of Homocysteine Lowering Folic Acid Improves Endothelial Function in Coronary Artery Disease via Mechanisms

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Influence of folic acid on plasma homocysteine levels & arterial endothelial function in patients with unstable angina.

BACKGROUND & OBJECTIVE High plasma homocysteine (Hcy) levels are known to be associated with coronary artery disease, but the precise level associated with an increased risk is yet controversial. Whether the beneficial effects of folic acid on arterial endothelial function persist over longer periods is not known. This study was carried out to assess whether folic acid supplementation could pro...

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Folic acid improves arterial endothelial function in adults with hyperhomocystinemia.

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Reduction of total homocysteine levels by oral folic acid fails to improve endothelial function in children with chronic renal failure.

tables or figures and should relate solely to an article published in Circulation within the preceding 12 weeks. Authors of letters selected for publication will receive prepublication proofs, and authors of the article cited in the letter will be invited to reply. Replies must be signed by all authors listed in the original publication. Please submit three typewritten, double-spaced copies of ...

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ژورنال

عنوان ژورنال: Molecular Medicine Reports

سال: 2014

ISSN: 1791-2997,1791-3004

DOI: 10.3892/mmr.2014.2332